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COVID-19 Promotes “Hyperactivity” in Blood-Coagulating Cells, Studies Show




COVID-19 (New Coronavirus Infection) (# If there is no character limit, add parentheses when first appearing Platelet-induced changes may contribute to the development of heart attacks and strokes in some patients, a new study reveals.

Scientists at the University of Utah state that inflammatory proteins produced during infection can significantly change the function of platelets, making them “hyperactive” and susceptible to the formation of dangerous and potentially fatal blood clots. I have found

Researchers hope that a better understanding of the causes of these changes will lead to more effective treatments for patients. Their report is blood, Journal of the American Society of Hematology.

Researchers say coronaviruses have a moderate prevalence in countries with early adoption of facemasks

“Our findings add an important part to a jigsaw puzzle called COVID-19,” said Robert A. Campbell, senior professor of research and assistant professor of internal medicine. statement.. “We found that infection-induced inflammation and systemic changes affect platelet function, causing platelets to aggregate faster, which explains why the number of blood clots is increasing in COVID patients. “

According to scientists, some evidence suggests that COVID-19 is associated with an increased risk of blood coagulation and in some patients may lead to other cardiovascular problems ..

Researchers investigated 41 COVID-19 patients admitted to a hospital in the University of Utah Salt Lake City. The blood of these patients was then compared to samples taken from healthy, age- and gender-matched individuals.

Impact of COVID-19 documented by satellite imagery on new initiatives from the US, Japan and Europe

Researchers have discovered that the virus that causes COVID-19, SARS-CoV-2, appears to induce genetic changes in platelets. These changes affect how platelets interact with the immune system, contribute to respiratory inflammation, and can cause more severe lung damage.

“There are genetic processes we can target that prevent the platelets from changing,” Campbell explained. “Understanding how COVID-19 interacts with megakaryocytes or platelets may block that interaction and reduce the risk of developing a blood clot.”

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