Health

The key to slowing aging may be in your genes, blood iron levels-more lifestyle

Group of scientist Genes associated with aging were identified. Studying genes helps explain why some people age at a different rate than others.

An international study using genetic data from more than one million people suggests that maintaining healthy levels of iron in the blood may be the key to improving aging and living longer doing.

The findings may accelerate the development of drugs that reduce age-related illnesses, prolong life well, and increase the chances of getting disease-free and aging, the researchers say.

Scientists at the University of Edinburgh and the Max Planck Institute for Aging Biology in Germany have focused on three measures related to biological aging-lifespan, disease-free (healthy), and very longevity (longevity). ).

Biological aging-the rate at which our body slows down over time-is different for each person and causes the world’s deadliest illnesses such as heart disease, dementia and cancer.

Researchers pooled information from three public datasets to enable unprecedented detailed analysis. The combined datasets were equivalent to studying 1.75 million lifespans or over 60,000 very long-lived people.

The team has identified 10 regions of the genome associated with longevity, healthy longevity, and longevity. They also found that the iron-related gene set was overexpressed in the analysis of all three measures of aging.

Researchers use this to statistically suggest that genes involved in blood iron metabolism are part of the cause of healthy longevity (known as Mendelian randomization). confirmed.

Blood iron is affected by diet, and abnormally high or low levels are associated with age-related conditions such as Parkinson’s disease, liver disease, and a reduced ability of the body to fight infection in the elderly.

Researchers say that designing drugs that can mimic the effects of genetic mutations on iron metabolism might be a future step to overcome some of the effects of aging, but more Please note that this requires work.

The study is funded by the Medical Research Council and published in the journal Nature Communications.

Anonymized datasets that relate genetic diversity to healthy lifespan, longevity, and longevity were downloaded from the publicly available Zenodo, Edinburgh DataShare, and Longevity Genomics servers.

Dr. Paul Timmers of the Usher Institute at the University of Edinburgh said: “These findings are very exciting because they strongly suggest that high levels of iron in the blood shorten our healthy lifespan. Damage from aging.

“We may begin to explain why our findings on iron metabolism are associated with very high levels of iron-rich red meat in the diet associated with age-related conditions such as heart disease. I guess it’s not possible,” Timmers added.

On the other hand, Dr. Joris Diren of the Max Planck Institute for Aging Biology, Germany, said: “Our ultimate goal is to discover how aging is regulated and find ways to improve health during aging. We have discovered lifespan, healthy lifespan, and lifespan-related genomes. All 10 areas of are exciting candidates for further research.”

(This article has been published from the telegraph agency feed with no changes to the text. Only the headings have changed.)

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