Health
Possible causes for long-term COVID-19
Researchers at Stanford University have released information to show that in inpatients1 With severe COVID-19, the virus can cause the production of autoantibodies, effectively attacking itself by the body and causing inflammatory diseases. Faculty of Immunology and Rheumatology, and the Institute of Immunology, Transplantation and Infectious Diseases, Stanford University School of Medicine, Stanford, California.
This finding may explain the long-term symptoms seen in some patients.
“This suggests that inflammation in response to SARS-CoV-2 infection may promote tissue damage in the acute phase and promote some of the long-term sequelae,” the authors said. rice field.
Immunoglobulin G (IgG) autoantibody measurement
Researchers have developed three protein arrays to measure IgG autoantibodies, anti-cytokine antibodies, and anti-viral antibody responses in serum samples associated with connective tissue disease.
The researchers also reported that autoantibodies were identified in about half of the 147 patients admitted with COVID-19, as opposed to less than 15% of 41 healthy controls.
When these autoantibodies are present, they primarily target self-antigens associated with myositis, systemic sclerosis, and duplication syndrome, a rare disorder.
However, a subset of traditional autoantigens or cytokine-targeted autoantibodies only develop after SARS-CoV-2 infection.
“Autoantibodies track the long-term development of IgG antibodies that recognize a subset of SARS-CoV-2 structural and non-structural proteins, but proteins from influenza, seasonal coronavirus, or other pathogenic viruses. We do not recognize that SARS-CoV-2 causes the development of newly developing IgG autoantibodies in a significant proportion of hospitalized COVID-19 patients, with a positive immune response to the SARS-CoV-2 protein. We conclude that there is a correlation, “the authors conclude.
Read more COVID-19 Coverage
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reference
1. Chang SE, Feng A, Meng W, etc. Newly developed IgG autoantibodies in inpatients with COVID-19. Nat Commun 2021; 12: 5417; https://doi.org/10.1038/s41467-021-25509-3
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