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Diagnostic bias in post-SARS-CoV-2 conditions

Diagnostic bias in post-SARS-CoV-2 conditions

 


Dear Editor,

I read with great interest the study by Ballouz et al. [1].

It should be emphasized that, apart from self-reported issues (honesty, symptom awareness), participants did not undergo diagnostic screening to accurately identify their illness or health condition.

Overall pre-infection health status and all factors that could exacerbate it were not monitored.Research reveals that symptoms similar to the new coronavirus already exist for various reasons other than the new coronavirus. [2]. Nutrient data are essential as they interact with mild systemic inflammation (SI).

Since SI is associated with cardiovascular disease, and novel coronavirus is a vascular disease, SARS-CoV-2 may only exacerbate existing SI. Metabolic and vascular diseases affect all tissues, but since SARS-CoV-2 is a vascular disease, a multisystem inflammatory response syndrome derived from it has been described. Each individual is unique and symptoms are affected by SI grade and overall vulnerability. Also, because vascular disease affects all tissues, symptoms can vary. To estimate the actual viral harm, all possible reasons for stimulating inflammation must be monitored.

Infections and other immunostimulatory conditions can cause a variety of medical conditions. Therefore, the state after SARS-CoV-2 infection may actually be the onset, or early stage, of a new disease. Individuals fail to recognize and assess the early onset of new illnesses, linking their symptoms to COVID-19. Moreover, relatively healthy subjects are healthier than those with any disease. In conclusion, infected cases need to be evaluated relative to previous medical conditions, rather than population-based samples labeled as healthy, in parallel with other factors that may influence inflammation. there is.

Apart from the erroneous test result, it was clear that he was a SARS-CoV-2 carrier and had foot symptoms.Suspect no history of infection in this scenario [3]. Antibody screening to confirm such a history is not performed in epidemiologic studies.

Finally, ‘after SARS-CoV-2 infection’ is better than ‘after COVID-19 infection’ because a positive test is only a carrier of a biomolecule.

References

1. Ballouz T, Menges D, Anagnostopoulos A, et al. Recovery and symptom trajectory up to 2 years after SARS-CoV-2 infection: a population-based longitudinal cohort study. BMJ 2023;381:e074425. Doi: 10.1136/bmj-2022-074425
2. Murio, DS, Kotsiu, OS, Gurugurianis, KI (2021). Estimation of COVID-19 risk factors and predictors of vulnerability to infection among social strata. International Journal of Environmental Research and Public Health, 18(16), 8701. https://doi.org/10.3390/ijerph18168701
3. Murio DS, Gruglianis KI. False-positive and false-negative his COVID-19 cases: respiratory prevention and control strategies, vaccination, and further perspectives. Expert Review of Respiratory Medicine 2021;15:993–1002. Doi: 10.1080/17476348.2021.1917389

Competing Interests: No Competing Interests

Sources

1/ https://Google.com/

2/ https://www.bmj.com/content/381/bmj-2022-074425/rr

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