Health
Self-segregation may increase vulnerability to coronavirus, studies reveal
Self-separation is the standard way to protect others and slow the spread of COVID-19. If you have or may have been infected with a virus, it is very important to limit the number of people you have in contact for 14 days.
This is the most effective way to prevent the spread of coronaviruses. RHowever, researchers now argue that the time spent in quarantine may actually increase vulnerability to upper respiratory tract viruses and possibly coronaviruses.
Daily mirror
“Some of us who were exposed to the coronavirus COVID-19 (new coronavirus infectious disease) (# If there is no character limit, add parentheses when first appearingThey are more likely to develop the disease than others.” Sheldon Cohen, a research author at Carnegie Mellon University in the US, said.
“But our research on psychological factors that predict susceptibility to other respiratory viruses may provide clues to help identify key factors in COVID-19,” Cohen added. It was.
Through a series of virus attack studies, we investigated how such factors affect whether healthy adults exposed to respiratory viruses become ill.
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Cohen’s study found that eight virus strains that cause colds (Rhinovirus types 2, 9, 14, 21, 39 and Hanks, respiratory syncytial virus and coronavirus 229E) and two virus strains that cause influenza (A/Kawasaki / 86) H1N1; and A / Texas / 36/91).
“Our research intentionally exposes people to the common cold and flu virus, and how psychological and social factors are effective for the immune system to control infection or prevent or mitigate the severity of disease. I researched whether to predict.”
Delay the spread of Coronavirus, Many countries have announced home-based measures, increasing interpersonal stressors such as loneliness, unemployment and family conflicts.
Cytech Daily
According to Cohen, these stressors could be strong predictors of how people respond when exposed to coronaviruses.
A series of studies found that participants experiencing interpersonal stressors were more likely to develop upper respiratory tract disease when exposed to the cold virus.
Cohen believes interpersonal stressors may play a similar role in response to causing the coronavirus COVID-19 (new coronavirus infectious disease) (# If there is no character limit, add parentheses when first appearing, Increase a person’s vulnerability to infections and illnesses.
In addition, both social and psychological stressors increased production of cytokines, molecules that promote inflammation in response to infection.
In Cohen’s study, psychological and social stressors were associated with overproduction of inflammatory cytokines in response to colds and influenza viruses. Second, this excess inflammation was associated with an increased risk of getting sick.
Similarly, studies on COVID-19 have shown that production of excess proinflammatory cytokines is associated with more severe COVID-19 infection, and an excess cytokine response triggered by stress It suggests the hypothesis that it may contribute to excessive inflammation and symptoms as well.
Cohen’s study points out the importance of social and psychological factors in the development of infection and disease. This work may hold clues to the health effects of ongoing quarantine.
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