COVID-19 risk does not grow with blood pressure medication

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A device used to test for high blood pressure.
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Risks associated with COVID-19 (New Coronavirus Infection) (# If there is no character limit, add parentheses when first appearing According to two research studies, it does not increase when taking blood pressure medications.

Research I saw Two different types of blood pressure medications, ACE inhibitors (drugs ending in -pril) and ARBS (drugs ending in -sartan). Brand names include lisinopril, captopril, losartan, balsatan, zestril, prinisil, cozar, atacand.

Hypertension is a widespread problem in the United States, Where the quote says One in three Americans suffers from high blood pressure so severe that it requires medication.

Here’s what you need to know.

According to one study, the risk of becoming infected with COVID-19 or, if infected, becoming severe with COVID-19 does not “substantially increase”

Study table

First study Problematic Renin-Angiotensin-Aldosterone Inhibitor and Covid-19 Risk Published in New England Medical Journal. Released May 1, 2020.

The researchers wrote:

Previous treatment with ACE inhibitors, angiotensin receptor blockers, beta blockers, calcium channel blockers, or thiazide diuretics and possible positive or negative Covid-19 test results, and potentially severe Of patients who tested positive (defined as intensive care, mechanical ventilation, or death).

They studied more than 12,000 patients.

Of the 12,594 patients tested for Covid-19, a total of 5894 (46.8%) were positive. 1002 of these patients (17.0%) were severe. There was a history of hypertension in 4357 patients (34.6%), of which 2573 (59.1%) were positive. 634 of these patients (24.6%) were severe. There was no association between a single drug class and an increased likelihood of positive tests. None of the tested drugs was associated with a significant increase in the risk of severe illness in patients who tested positive.

The study concluded that:

In this study, patients were examined in the “New York University (NYU) Langone Health Electronic Medical Record”.

A second study found no difference in the severity, complications, or risk of COVID-19 in patients taking some hypertensive drugs

GettyThis photo from the Center for Disease Control and Prevention (CDC) shows a microscopic image of coronavirus.

Second study Was called Association of Renin-Angiotensin System Inhibitors with Severity or Risk of Death in Hypertensive Patients Hospitalized for Coronavirus Disease 2019 (COVID-19) Infection in Wuhan, China. It was published in the JAMA Cardiology journal in late April 2020.

This study tried to answer the questions:

Among patients with hypertension, those taking angiotensin-converting enzyme inhibitor (ACEI) or angiotensin receptor blocker (ARB) were more severe at the time of hospitalization due to coronavirus disease 2019 (COVID-19) infection. Or is the risk of death higher?

This study included 362 patients with hypertension hospitalized for COVID-19 infection.

What is the conclusion? “People taking ACEIs / ARBs did not differ in disease severity, complications, or risk of death compared to those not receiving these medications.”

Researchers add: These results support current guidelines and social recommendations for treating high blood pressure. “

According to the New York Times, Studies are also investigating whether antihypertensive drugs reduce the risk of COVID-19.

Medical groups do not suggest that people discontinue their hypertensive medications during a pandemic.

American Heart Association Previously issued a statement “Currently shows beneficial or adverse outcomes from background use of ACE inhibitors, ARBs or other RAAS antagonists in COVID-19 patients with a history of cardiovascular disease treated with COVID-19 or such agents There are no or clinical data. “

Tissues are currently prescribed such drugs for the indications that HFSA, ACC, and AHA are “beneficial for these drugs such as heart failure, hypertension, or ischemic heart disease.” We noted that we recommend that patients continue with RAAS antagonists. “

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