Health
Common BP Drugs Do Not Increase COVID-19 Severity: Study
Scientists, including researchers at Northwestern University in the US, say drugs (ACE inhibitors and angiotensin receptor blockers (ARBs)) may cause an increase in the human cell surface receptor protein ACE2 used by the novel coronavirus There is widespread concern that there are invading cells and infecting them.
Earlier studies warned that this class of drugs could increase the risk of new coronavirus SARS-CoV-2 infection and increase the severity of COVID-19.
However, new findings published in the Journal of the American Society of Nephrology revealed that ACE2 in the renal membranes of mice was decreased rather than increased, and lung membranes remained unchanged.
According to the researchers, this study was the first to study the effects of ACE2 and ARB on the lungs, considered to be the main target of SARS-CoV-2 entry into the body, and Supports safety. Pandemic face.
“This study supports the notion that the use of ACE inhibitors and angiotensin receptor blockers does not increase the risk of COVID-19 infection,” said Daniel, co-author of a study at Northwestern University. Battle said.
Scientists have said ACE inhibitors and ARBs are a category of drugs called RAS blockers.
They said that these drugs block the action of small proteins that cause blood vessel narrowing and fluid retention by the kidneys and raise blood pressure by different mechanisms.
Studies show that these drugs help relax or dilate blood vessels and reduce fluid retention. Both reduce blood pressure.
In the current study, scientists isolated the kidney and lung membranes of mice treated with either the widely used ACE inhibitor captopril or the widely prescribed ARB telmisartan. Measured ACE2.
Since ACE2 was recognized as the major receptor used by the virus to enter host cells, scientists have identified the potential for susceptibility and worse clinical course of COVID-19 in patients treated with these agents. He said there were multiple studies discussing risk.
They said much of the speculation came from previous animal studies where there was a RAS blocker that was reported to increase ACE2 production in heart and kidney vascular cells.
“We weren’t biased in any way, and the finding of kidneys with lower ACE2 in treated animals was a bit unexpected,” said Jan Wysocki, the first author of the study. PTI VIS VIS VIS
Disclaimer:- This article has not been edited by Outlook staff and is auto-generated from the news feed. Source: PTI
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