Health
Coronavirus: U.S. research identifies genes that fight viral infections
A new study has been published to identify human genes that help fight the Toronto-Coronavirus infection. This can be the first step in finding new treatment options for patients with severe COVID-19.
Researchers at Sanford Burnham Pre-Beads in California Researched on molecular cells last Tuesday Identify 65 genes that control replication of the SARS-CoV-2 virus, the virus that causes COVID-19.
These genes are called interferon-stimulating genes and are associated with interferon, a protein produced by the body to fight the virus.
“We wanted to better understand the cellular response to SARS-CoV-2, including those that cause strong or weak responses to infection,” said lead author Sumit K. Chanda. In a news release.. “We have gained new insights into how the virus invades human cells, but we are looking for the Achilles tendon so that we can develop the best antiviral agents.”
In addition to SARS-CoV-2, some of these genes have been shown to affect many unrelated viruses such as seasonal influenza, West Nile fever, and HIV.
One of the notable human genes identified is BST2, which is called “a potent inhibitor of SARS-CoV-2 replication” in this study. And expression of this gene was found to reduce virus replication by 53% and virus release by 74% when compared to control cells.
Researchers relied on knowledge gained from research on the coronavirus SARS-CoV-1, which caused the fatal outbreak of SARS between 2002 and 2004.
Of the five or more recently discovered genes, eight were found to inhibit both SARS-CoV-2 and SARS-CoV-1.
“This is important information, but we need to learn more about viral biology,” research author Laura Martin Sancho said in a news release about whether variability within these genes correlates. He also pointed out that further research is needed. COVID-19 severity.
Chanda’s team is currently investigating a variant of SARS-CoV-2 of concern.
“Now that vaccines are helping to control pandemics, it’s very important that we stay on top of our basic research efforts,” said Chanda.
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